Endocrine

Infection with SARS-CoV-2 can cause pancreatic impairment (2024)

Our molecular pathology results and multi-omics data suggest that SARS-CoV-2 infection intensifies pancreatic lesions and heightens the risk of diabetes in the elderly.

In considering the heightened risk of insulin-dependent diabetes in the elderly following COVID-19, our attention is drawn to the β cells. Based on our pathophysiological findings, we posit that SARS-CoV-2 infection may harm β cells in older models via several mechanisms.

Risk for newly diagnosed diabetes after COVID-19: a systematic review and meta-analysis (2022)

Our subgroup analysis revealed a 1.2-fold increased risk of developing diabetes after COVID-19 compared to patients with other upper respiratory tract infections and a 1.82-fold increased risk of developing diabetes after COVID-19 compared to the general population.

Risks and burdens of incident diabetes in long COVID: a cohort study (2022)

Risks and burdens of post-acute COVID-19 diabetes outcomes by severity of the acute infection compared with the contemporary control group, from The Lancet Diabetes & Endocrinology.